October 31, 2012
STAGE EDUCATEUR ET MONITEUR CANIN NOVEMBRE
STAGE EDUCATEUR ET MONITEUR CANIN NOVEMBRE
> STAGE 1er Degré MONITEUR CANIN
Date stage: 3 et 4 novembre 2012- LAON (02)
Date examen: 23 décembre 2012
> STAGE 1er Degré MONITEUR CANIN
Date stage: 3 et 4 novembre 2012- LAON (02)
Date examen: 23 décembre 2012
> Stage 1er degré MONITEUR CANIN
Date stage: 24 et 25 novembre 2012 - SAINT BRIEUC (22)
Date examen: 16 et 17 février 2013
Pour plus d'informations :
http://magazinecneac.fr/?q=groupeT%2Fformation%2F1degre-stages-2012&page=1
Date stage: 24 et 25 novembre 2012 - SAINT BRIEUC (22)
Date examen: 16 et 17 février 2013
Pour plus d'informations :
http://magazinecneac.fr/?q=groupeT%2Fformation%2F1degre-stages-2012&page=1
October 30, 2012
Potential causes of reproduction problems
Things to look into if you are having trouble with any of the following: infertility, reabsorption of fetuses, weak/sick puppies, birth defects, etc.
1) Water- especially well water can be high in nitrates, copper, lead and other toxic substances. Have water tested or try using bottled water instead.
2) Infections such as brucellosis, beta
strep, mycoplasma, toxoplasmosis, herpes, parvovirus etc. Not all are susceptible to Clavamox or other common antibiotics so don’t rule out infection based only on an unsuccessful trial of one antibiotic. (For example, Clavamox is effective against beta strep, but not against Mycoplasma.) ANY infection (tooth infection, upper respiratory infection, etc) in the dam that causes a fever in the first trimester may cause birth defects or loss of the litter.
3) Diet- lack of folic acid, excess vitamin A (found in high amounts in liver), copper and other dietary imbalances. Flaxseed has been indicated by some (may act as an estrogen antagonist). Phytoestrogens in soy can also impact reproductive health. Some mushrooms and plants may be toxic, moldy foods that can be found in the yard, some nuts, etc. Tomato pomace may contain higher amounts of pesticide residue than other fiber sources.
4) Pesticides- Nearby farms may be spraying crops/ your neighbors might be spraying pesticides in the yard which the wind is carrying over, the park you walk your dog in might be treated with pesticides.
5) Lack of sunlight
6) Cleaning chemicals- One person had trouble with infertility and puppies dying from using Tide laundry detergent in water to clean wood enclosures. Pinesol has been implicated by some. There are many more that could potentially cause issues if absorbed through the paw pads or licked. Vinegar and water or diluted bleach tend to be safe for cleaning.
7) Flea, tick, ear mite and mange chemicals
8) Medications- steroids are one example. Also, topical retin A preparations used for acne on owner’s skin may cause birth defects in pups if pregnant bitch comes in contact with it. Be careful with any topical medications used (especially if containing retin A/ retinol- also included in some over-the-counter anti-aging creams) as well as birth control patches, menopause estrogen therapy patches, use of insulin, etc.
9) Metabolic imbalance in dam- hypothyroid, diabetes, etc.
10)Mold- in food or wood that is chewed on, nutshells, etc.
11)Vaccinating close to or during pregnancy
12) Paint or wood protector that is ingested from wood fencing or enclosures. Also compressed wood used to make cheap furniture.
13) Second hand smoke (implicated in premature birth and low birth weight of puppies)
October 29, 2012
Sharon Redmer
The Officers and Board of Directors of the American Belgian Tervuren Club is proud to share that the winner of the AKC Lifetime Achievement Award (Companion Events) is Sharon Redmer, StarBright Belgian Tervuren. Congratulations, Sharon!http://www.akc.org/news/lifetime/2013/recipients.cfm
Companion Events: Sharon Redmer
Sharon began training her first dog, an All-American shelter rescue, with the Ann Arbor Dog Training Club in 1970. Her first Belgian Tervuren, acquired in 1972, became a multi-High-in-Trial UDT. For over 30 years, Sharon has pursued two passions: being the very best AKC judge she can be and breeding beautiful Belgian Tervuren with the character to compete in every venue.
Over 150 of her "StarBright" bred Tervuren have earned CHs, GCHs, OTCHs, MACHs, HTCHs, and CTs plus many obedience, agility, herding and tracking titles, Group and Specialty wins, High-in-Trials and perfect 200 scores.
Even judging 50 AKC shows a year, Sharon finds time to mentor new and hopeful obedience judges. Exhibitors in her ring find a judge with a welcoming smile and encouragement. Sharon’s philosophy is simple: "Kindness is never wasted."
A judge of the Herding Group, Poodles, Dobermans, Rally and Juniors, Sharon has officiated at past AKC National Obedience Invitationals including the inaugural Juniors’ competition in 2011. She has judged Nationals for her Belgian breeds (multiple times) and specialties on four continents.
An Honorary Life member of the Ann Arbor Dog Training Club and the Ann Arbor Kennel Club, Sharon has served the American Belgian Tervuren Club as President and AKC Delegate, and is a popular seminar presenter on judging the Tervuren. Sharon recently retired as the director for community outreach services at the University of Michigan Health System.
Constancy and Repeatability in Hip Evaluations
Constancy and Repeatability in Hip Evaluations
|
.
A magazine, newsletter, and website article by Fred Lanting
July 2000
|
Some questions have been raised about how long the PennHIP reading, and the two-year-old OFA (or one-year old "a" stamp, OVC, or GDC) results are good for.
The terms “precision” and “accuracy” may be used by your PennHIP vet. What he means by the former is that there is general agreement between “scrutineers/readers” as to the diagnosis; what he means about the latter is relative to additional factors. Imagine a sharpshooter’s bull’s-eye target, and half a dozen riflemen who shoot at it. All of their bullet holes are clustered somewhere in the upper left quadrant of the target, some distance from the bull’s-eye but all about 2 inches from each other. The people at Penn liken that to the hip-extended evaluators who agree fairly well, but miss many cases of HD with that position. Now imagine equally talented shooters with better rifles (scopes, longer barrel, etc.) who not only cluster their holes in the bull’s-eye (“accurate”), but also within one-sixteenth inch of each other (precise). Further, imagine those with the better guns being able to repeat their performance at every match (reliable). Accuracy, however, is not well defined in the context of making genetic change toward better hips. For that, you need to add the effect of heritability. The hip phenotype (as most accurately reflected in DI and percentile scores) with the highest heritability is the one that should be considered most accurate. And the distraction method has a much higher heritability than older methods of viewing hips.
The claim by Penn that OFA is not the best method breeders have for progress in reducing HD in their line or breed involves the accuracy factor. They call our attention to the fact that there are many dogs (usually of certain breeds) that do not develop DJD but are OFA-assessed as dysplastic because of laxity at two years’ age. Even more importantly, there is the greater number that were adjudged “normal” at two years but later developed DJD or, if not re-radiographed, produced an unacceptably high percentage of dysplastic descendants. This led to the conclusion that the accuracy of OFA’s method is gravely flawed. Even if reliability (by this is usually meant repeatability) were high from younger ages up to the two-year qualification age for OFA certification, and I do not think it is, the absence of accuracy is worrisome to breeders, and diminishes the importance of published reliability figures in 1997.
Remember the difference between reliability and accuracy, described above. As an example, Penn cites the 1996 OFA-type JAAHA evaluation of military dogs in a longitudinal study in which all the dogs with “normal” hips at two years had mild degenerative changes by nine years of age. At the same time, 22 of 52 dogs that had been judged “positive” for HD at two had similar changes by nine years! The conclusion is that the OFA-type evaluation at two years does gives a relatively high rate of misdiagnosis, and blurs the distinction between true positive-for-HD and true negative (no HD) diagnoses even at the supposedly “safe” age of two years. Admittedly, the OFA hedges its emphasis on laxity a little by using the phrase “normal for age and breed” when grading radiographs; they do allow for some differences between Saint Bernards’ and Borzois’ hips this way, though it is still a totally subjective evaluation. And the point that some people who wrote to me wanted explained was about OFA-Normals (at 2 yrs) going bad later in life. Read that study: Banfield CM, Bartels JE, Hudson J. A retrospective study of canine hip dysplasia in 116 military working dogs; Angle measurements and OFA grading. J Am Anim Hosp Assoc. 1996; 32:413-22. Other reading material for you: Corley EA, Keller GC, et al. Reliability of early radiographic evaluations for canine hip dysplasia obtained from the standard ventrodorsal radiographic projection. JAVMA 1997;211:1142-1146. But along with that you should also read the letter to the editor on p. 487-488 in JAVMA's Volume 212 of Feb. 15, 1998 . That letter corrects some misconceptions or possible misinterpretations.
Comparitively, the actual testing error of PennHIP (as determined by repeated tests of the same dog at one point in time) is extremely small (<0.05 DI units). The biological variability of a given dog’s hip laxity over time, however, could be much larger. There is variation in anything biological, one example being the evaluation of hip laxity, taken and scored such-and-such on a given day, and then on some future day scored so-and-so.
Consider three points. First, biological variability is evident in all measured biological parameters, e.g., serum cholesterol, blood pressure, heart rate, etc., even hip laxity to a smaller degree. Second, if a breeder believes the naysayers and feels distraction radiography should not be used because of the perception of too much variability (error), he should realize that in all studies that compared the DI with OFA score, the OFA diagnostic test was found to have even more error when evaluated longitudinally. The data is clear on this issue. Many people have been lulled into believing that since their dogs receive one OFA score at 2 years of age, that the score is absolute and will not change. If they would take the time, and spend the money to have repeated OFA testing done, they would find a troubling amount of error, and more error than with PennHIP.
Third, to circumvent the potential uncertainties occasioned by inherent biological variation (using any diagnostic test), it is wise to average many observations rather than fully rely on any single observation. Multiple tests will regress to the mean, giving a truer measure of the phenotype, the same way you will get closer to 50% “heads” the more often you flip a coin, or approach 12.5% “monorchids” the more often you breed two “carriers”. In other words, if a breeder is arguing that PennHIP should not be used because it has too much “error”, the OFA method should also be abandoned because its error has been shown to be even greater. Complaining because a biological situation does not have mathematical precision is simply wrong-headed. By the way, if there were any differences in reading DI at different times in a dog’s life, I would suspect that the “margin of error” probably is greater in the higher end of the scale. It has been my observation that the looser the joint, the greater variation in readings.
October 28, 2012
-Lament of a Stud Dog-
My job is making puppies, and I get two tries at that.
They pat me on the head and say "good boy"--and that's that.
It's half my job to give 'em teeth and toplines, fronts, and other.
Remember, it's only half my job. They also have a mother.
It's not my job to carry pups and make 'em grow and nurse 'em,
And feed and clean and make 'em strong, that's for their mother and a person. It's not my job to wean and feed the calcium and the food.
And stack and gait and housebreak and make 'em a showing brood.
It's not my job to plan the breeding and learn what produces well.
To study pedigrees, learn what's there, and pick out what to sell.
It's not my job to deliver a winner, it's only genes I sell.
But let the puppies turnout bad and guess who catches hell!
-Author Unknown
They pat me on the head and say "good boy"--and that's that.
It's half my job to give 'em teeth and toplines, fronts, and other.
Remember, it's only half my job. They also have a mother.
It's not my job to carry pups and make 'em grow and nurse 'em,
And feed and clean and make 'em strong, that's for their mother and a person. It's not my job to wean and feed the calcium and the food.
And stack and gait and housebreak and make 'em a showing brood.
It's not my job to plan the breeding and learn what produces well.
To study pedigrees, learn what's there, and pick out what to sell.
It's not my job to deliver a winner, it's only genes I sell.
But let the puppies turnout bad and guess who catches hell!
-Author Unknown
October 27, 2012
Frozen Semen AKC Protocol 2012
Frozen Semen AKC Protocol 2012
Many breeders in the USA purchase frozen semen for future use. This protocol is for that situation.
You will need to get a letter of transfer of ownership of the semen from the current dog frozen semen owner. This is not a form letter AKC has, it’s a letter that is written specifically from the dog frozen semen o
wner to you – the letter will state (key word) Transfer of Ownership of Frozen Semen – In this letter you will have the dog name & registration number, and the letter will basically state that the owner has transferred however amount of semen (number of vials) and on the date of collection to you (your name) and the owner will sign this letter. This letter will be given to the facility where you will have the semen stored. My suggestion is to have this letter done in triplicate and notarized, 1 copy to the facility-1 copy for your files-1 copy back to the frozen semen seller.
This facility will then send AKC a collection statement showing your name as being the owner of this particular amount of semen and will AKC will put this collection statement in the dog file and each and every time you send AKC a frozen semen litter application we pull this collection statement to see who the semen owner is and also AKC will subtract the number of breeding units being used for that particular breeding. This letter will authorize you as being the owner of that particular amount of semen to AKC, and you will then be the one to sign the frozen semen litter application that is all needed since this dog have a DNA nothing else is needed.
The AKC representative, if you have any questions is Sheila Hunter.
Her contact information follows:
Shelia Hunter
American Kennel Club
Sr. Registration Operator
Registration, Foreign & AI Department
Email: SJH@akc.org
919-816-3732(Direct) 919-816-4235(Fax)
October 26, 2012
AMSTERDAM WINNER SHOW 2012 - JUDGES
Belgian Shepherd Dog, Groenendael (15) | Sandra Dubach (The Netherlands) | Sunday |
Belgian Shepherd Dog, Laekenois (15) | John Wauben (The Netherlands) | Sunday |
Belgian Shepherd Dog, Malinois (15) | John Wauben (The Netherlands) | Sunday |
Belgian Shepherd Dog, Tervueren (15) | Lisbeth Mach (Zwitserland) | Sunday |
October 25, 2012
Premature Puppy Protocol
Premature Puppy Protocol - Myra Savant-Harris
written 2009
I've been teaching this information at seminars lately and I've spent the last couple of months researching it to the best of my ability to have it ready to present to you. Researching this stuff is often extremely difficult because there are so few written references to it, so I first have to review everything I know about human medicine and then loosely translate it to dog medicine......without being a vet. It's often difficult. If you think it will be useful, please print it up and keep it among your whelping supplies. Don't expect that every vet would be familiar with this protocol, and don't think you will find it written somewhere else, because you won't....maybe bits and pieces but not all connected in protocol form. I doubt that more than a handful of vets will be familiar with these techniques in their entirety. I would like to take a couple of minutes to give you some background information so that you can understand how all the pieces fit together. My gift is called: Premature Puppy Protocol I hope that you will learn it as you have the Fading Puppy Protocol and be prepared should you have a premature puppy in the future. You will, again, need to get the meds from your vet and be prepared. The knowledge alone will not help you. You will need supplies on hand and ready. Even an extra hour or so may well lead to the death of your puppy.
In human OB for the last 20 years or so, we have administered a medication called Betamethasone (and some vets have also used Dexamethasone with the same results) to moms who were in premature labor that was not well controlled by Terbutaline or other medications. It is commonly ordered now by the doc and administered by nurses. It is safe. In human moms, it is given every 12 hours directly to the mother where it then crosses over the placental barrier to the baby to assist the baby to form surfactant in his lungs. Remember that the premature lungs are sort of like sticky little tubes. The baby has to work awfully hard to get air into those sticky little air tubes. Surfactant will change the surface of the sticky tissue so that instead of being sticky, it will be more slick....more soapy in "feel" so that the baby can breathe easily. If you know you are doing a planned c-section a day or two early, by all means, please ask your vet to administer Betamethasone to your bitch at least 12 to 24 hours prior to the surgery. He can administer it every 12 hours if he has time. He will give it by an intramuscular injection but in OB, it is given IV. Remember something......the human placenta is much more permeable than the canine placenta and much much much more permeable than the equine placenta, so all things being equal in the injection of the medication, a differing amount will reach the baby of each specie simply because of the placenta barrier. You may need to give the puppy (or other mammal) a dose of the betamethasone directly in order to maximize the production of surfactant even if mom was given the med. The main thing that you will accomplish when you administer surfactant is that the baby will then be able to coordinate his suck, swallow and breath reflexes.
If you have an unexpectedly early premature litter at home....you need to be prepared to care for the premature puppy. You will need a very small amount of BETAMETHASONE (or DEX) and IV fluids. Doctor Greer likes Normal Saline and for a premie, I believe that Normal Saline is the best fluid for sub cutaneous hydration. (I still like the potassium and sodium and calcium of lactated ringers for other uses, but for a premie......don't muddy the waters with anything but normal saline) You will need syringes and short needles.....preferable about a 23 gauge, 3/4 inch needle. If your bitch delivers puppies significantly early they will have no fur on their legs and muzzles, possibly extending back even further onto their heads. The legs maybe so hairless as to appear to be pink. The body may show the "markings" of the fur which will grow in, but only finer hair may be present. The face will be almost completely bald. They may or may not be smaller than normal puppies, but the bulk of them will be certainly be smaller than normal. You may have an occasional good sized puppy who is hairless and obviously a premature puppy. It does not matter how large he is....if he is a premie, he will still be struggling with the premature puppy problems of breathing, body temp and nursing.
As soon as possible....don't waste time.....administer betamethasone in a tiny, tiny dose to the puppy. Doctor Hutch has used the expression: Only let the Betamethasone 'talk' to the puppy. I always say: use a whisper of the drug. You are talking drops here. drops.
1. Weigh the puppy.
2. Record the Weight
3. Mix the Normal Saline and Beta Methasone using this formula:
4. One drop of Betamethasone for every 2 ccs of (WARMED to 98 degrees) normal saline
2cc.......1 drop
4cc.......2 drops
6 cc......3 drops
8 cc......4 drops
10cc......5 drops and so on.
Mix enough of the Saline plus Betamethasone to be able to administer the weight of the puppy in ccs sub cutaneously. If the puppy isn't nursing after 12 hours, give it again. Best way to warm is in your bra. Mix several ccs and just keep it tucked in your bra in case you need a second dose. Stop when baby has gained his full suck reflex and mom has milk.
1-2 oz puppy: Administer 1-2 cc of Normal Saline containing 1 drop of Beta
3-4 oz puppy: Administer 3 to 4 cc of normal saline containing 2 drops of Beta
and on and on
I wish I could answer questions like: How big a drop? Unfortunately, this is such an inexact science for me at this point, that I can't. There is a better way to mix it, but I would need to know the exact strength of the beta you are using to do that, so....just stick with the one drop per every two ccs of fluid. A little too much isn't going to hurt the puppy.....a little less may well simply not be enough. Err on the side of a big drop, ok?
The goal of the Betamethasone is to assist the puppy's body to manufacture surfactant which will enable the puppy to be able to suck, swallow and breathe at the same time. This will allow the puppy to breast feed. Over the last months, I've talked to a breeder in Phoenix who was able to keep a total of 5 tiny puppies weighing around 1.5 oz alive and nursing by using this technique. I wish I had gotten her name, but I didn't remember to do that. She was instructed in this technique by Doctor Hutch. I have refined and expanded it somewhat to include the human med aspect of it.
You must be prepared to oxygenate the puppy with oxygen running at no more than 1 liter per minute as his lungs mature.
An explanation of the warmth you will need:
A normal, full term puppy has a normal body temp of 95-96 degrees. A premature puppy, however, has left the "oven" (100-101 at time of delivery) too early; before the cookie is cooked, so to speak. He isn't ready to be at 95-96 degrees. He is an "unbaked" cookie. He needs to continue the baking process until the day arrives for his correct delivery date. His temp needs to be around 100 degrees and stable. (once the puppy is stable, you can begin decreasing the temp about 1/2 degree every day until by delivery day, he should be good at 95-96 degrees) Use a heating pad set at 100 degrees and use a rheostat (available at Pet Smart and used for Reptiles) to set his warming pad temp at 100 degrees. You will need a hot box....Isolette...for your premies. Do not give a premature puppy to mom except to nurse.....and not until he has a suck reflex. The puppy's body can be loosely wrapped in Saran Wrap to help him to maintain his body temp as long as he doesn't wiggle out of it and get it around his face. He can be covered with a loose cloth if he is moving around. Wrap him a warm piece of flannel while he is in the box if his temp appears to vary from exam to exam. Check him often for warmth. The premie will be much more difficult to manage as far as body temp goes. He will not be able to hold onto body heat for long, so for feedings, you will need to wrap his warmed body in saran wrap and cover with a cloth and hold him up to the mom's breasts for feeding.
You may need to use a breast pump to stimulate mom's breast milk to come in, and give her Fenugreek to encourage milk production.
Continue using subQ hydration on the puppy every 4 hours.....his weight in WARMED IV fluids for the first 12 hours. Place a small amount of glucose source: karo, honey, or tubed decorator frosting on his tongue within minutes after birth and every 3-4 hours after that. After 12 hours of age, If the puppy is stable, but still unable to suck, you will need to tube him. Use a WARMED formula of your choice and begin oral feedings by tube feeding. Feed him half his weight in ccs every 2 hours. DO NOT SYRINGE FOOD INTO THE MOUTH OF A PUPPY. You will kill a premie by syringing for a certainty. They cannot yet coordinate a suck swallow reflex. You will actually end up eventually causing aspiration pneumonia on any puppy that you syringe, but for those of you who choose to ignore this......for sure you'll kill a premie and it won't take you long to do it. I wish I had a nickle for every time a breeder has told me: I syringed in formula, but he died anyway. No, he didn't die anyway, you killed him. He isn't equipped to have thin liquids put into the front of his mouth. He is designed to pull a nipple far back in his mouth up against his soft palate and suck. The next best alternative is to tube feed him until the Betamethasone has allowed him to develop his lungs sufficiently to suck, swallow and breath.
Read this over a few times before you ask questions if you have them. Give it time to sink in.
October 24, 2012
October 23, 2012
Exposition de Varese 2012
Exposition de Varese
Dans le cadre de l'Alliance Cynophile Latine, se déroule les 3 et 4 novembre 2012 à Varese (Italie), l'Insubria Winner 2012 avec attribution du CACS de la SCC
Pour plus d'informations :
http://www.scc.asso.fr/
October 22, 2012
Metz 2012
WEETONEON ARTIC STORM, Puli Blanc, a été sacré Meilleur chien de l’exposition.
La Réserve de BIS est allée à EMBEREZ THE BIG SHOW, Dogue de Bordeaux devant SLICEY WHISKEY IN THE JAR Lakeland Terrier, 3ème meilleur chien de l’exposition.
Le juge était le Suisse Laurent Pichard.
courtesy: SCC
Removing the Stigma of Genetic Disease
Removing the Stigma of Genetic Disease
First published in the October, 2003 AKC Gazette
By Jerold S Bell, DVM
First published in the October, 2003 AKC Gazette
By Jerold S Bell, DVM
An inevitable consequence of breeding is the occurrence of genetic problems. No one wants to produce affected dogs, yet some breeders and owners are quick to assign blame. There are no perfect dogs, and all dogs carry some detrimental genes.
The emotional reaction to producing a dog with a genetic disorder often follows what is called the grief cycle:
· Denial: This isn't genetic. It was caused by something else.
· Anger: This isn't right! Why is this happening to my dogs?
· Bargaining: My dog sired more than 100 other dogs that are healthy. So this one doesn't really count, right?
· Depression: My kennel name is ruined. No one will breed to my dogs.
· And, finally, acceptance: My dog was dealt a bad genetic hand. There are ways to manage genetic disorders, breed away from this, and work toward a healthier breed.
Getting beyond denial
Unfortunately, many breeders can’t get beyond the denial stage. Some will hold to increasingly improbable excuses, rather than accept that a condition is genetic. They will falsely blame relatively rare disorders on common viruses, bacteria, or medications. The fact that these organisms or drugs are common to millions of dogs annually that do not have these disorders is not considered.
Some owners state that their veterinarian recommended not sending in a hip radiograph because the dog would probably not get certified. Then these owners lull themselves into believing that since the dog wasn't evaluated, it does not have hip dysplasia. The fact that a dog does not have an official diagnosis does not mean the dog is normal or "not affected."
It is important to confirm diagnoses of genetic disorders with blood tests, radiographs, or pathology specimens. However, the primary concern should always be for the individual dog. If an affected dog is not suffering, it should not be euthanized simply to obtain a pathological diagnosis. The increased availability of non-invasive techniques has made diagnoses easier to obtain.
Once confirmation of a genetic disorder is made, denial sometimes becomes deception, which is not acceptable. There are breeders who actively seek to prevent diagnoses and later necropsies, but who eventually realize that their actions are detrimental to their breed, and in the long run to themselves.
Working together to improve our breeds
Reducing the stigma of genetic disease involves raising the level of conversation from gossip to constructive communication. Dealing with genetic disorders is a community effort. Each breeder and owner will have a different level of risk or involvement for a disorder. We do not get to choose the problems we have to deal with. Breeders should be supportive of others who are making a conscientious effort to continue breeding their dogs while decreasing the risk of passing on defective genes.
Breeders ought to follow up on the puppies they have placed. They should periodically contact their buyers and ask about the health of the dogs. Some breeders fear they will be castigated if a dog they placed develops a problem. However, the vast majority of owners of affected dogs are pleased that their breeder is interested in their dog, and in improving the health of the breed so that other affected dogs are not produced.
A breeder cannot predict or prevent every health problem. If an owner's dog is discovered to have a problem, show your concern.
Breeders and breed clubs should be cooperative and supportive of researchers studying genetic disorders in their breed. Through research funded by breed clubs and by the AKC Canine Health Foundation (CHF), new genetic tests for carriers of defective genes are continually being developed.
The Canine Health Information Center was established by the CHF and the Orthopedic Foundation for Animals. CHIC is an online registry that works with the breed parent clubs to establish a panel of testable genetic disorders that should be screened for in each breed. The beauty of the CHIC concept is that dogs achieve CHIC certification by completing the health-checks. Passing each health test is not a requirement for certification. CHIC is about being health conscious, not about being faultless.
My hope for each breed is that there will eventually be so many testable defective genes that it will not be possible for any dog to be considered "perfect." Then we can put emotions aside and all work together on improving our breeds.
Breeders must lead the way to remove the stigma of genetic disorders. The applications for both the OFA and CHIC health registries include options that allow for open disclosure of all health-test results or semi-open disclosure listing only normal results. It is up to breeders to show that we are ready to move genetic disorders out of the shadows and check off the boxes for full disclosure.
More national clubs are having health seminars and screening clinics at their specialties. It was thought these events would scare away potential owners. We now know that without addressing the problems, in the long run, the breed may not be there for the owners.
October 21, 2012
Exposicion Internationale POITIERS - Speciale - full BSD results
Chien de berger belge Groenendael Classe INTERMEDIAIRE MALE Juge : Mme VARLET Marie-France |
1 | GHOST DE LA FORET D'OLIFAN | 2e | EXCELLENT | |
2 | JEDAIBLACK DE LA MAISON DU BOIS | 1er | EXCELLENT RCACS - RCACIB |
Chien de berger belge Groenendael Classe OUVERTE MALE Juge : Mme VARLET Marie-France |
3 | DATSUNDE DE LA FORET D'OLIFAN | - | ABSENT | |
4 | DIXON DU VERGER DE LA GROUE | 2e | EXCELLENT | |
5 | ECKMUL DE KENATIER | 1er | EXCELLENT CACS - CACIB | Meilleur de Race |
6 | EVEN DU PRE DE L'AUBOIS | 3e | EXCELLENT | |
7 | FASTOCHE DES LOUPS D'ANNE DE BRETAGNE | 4e | EXCELLENT |
Chien de berger belge Groenendael Classe JEUNE MALE Juge : Mme VARLET Marie-France |
8 | GROOVE DU BOIS DU TOT | 1er | EXCELLENT | |
9 | HICARE DE L'EMPREINTE DE TOUNDRAL | - | TRES BON |
Chien de berger belge Groenendael Classe VETERAN MALE Juge : Mme VARLET Marie-France |
10 | VIGOR DE CONDIVICNUM | 1er | EXCELLENT |
Chien de berger belge Groenendael Classe OUVERTE FEMELLES Juge : Mme VARLET Marie-France |
12 | CANDY BLACK DU PRE DE L'AUBOIS | 2e | EXCELLENT RCACIB | |
13 | DEE'GYN DU DOMAINE DE LA NOBLERIE | 3e | EXCELLENT | |
14 | EASY-LOVE DU PERIGORD VERT | 4e | EXCELLENT | |
15 | ERHA DU PRE DE L'AUBOIS | - | ABSENT | |
16 | FEELYA DU DOMAINE DE LA NOBLERIE | 1er | EXCELLENT CACS - CACIB |
Chien de berger belge Groenendael Classe JEUNE FEMELLES Juge : Mme VARLET Marie-France |
17 | HELSY DE L'EMPREINTE DE TOUNDRAL | - | ASSEZ BON | |
18 | HYDILLE DE L'EMPREINTE DE TOUNDRAL | - | TRES BON |
Chien de berger belge Malinois Classe INTERMEDIAIRE MALE Juge : Mme VARLET Marie-France |
20 | GALZOU DE LA CITE DES LOUVES | 1er | EXCELLENT | |
21 | GOLIATH DES ILES DU SUD | 2e | EXCELLENT |
Chien de berger belge Malinois Classe OUVERTE MALE Juge : Mme VARLET Marie-France |
22 | CALI DU DOMAINE DE VAUROUX | 1er | EXCELLENT RCACS - RCACIB | |
23 | CYSKO DU MAS DES LAVANDES | 2e | EXCELLENT | |
24 | DJAGGER DES ILES FIDJI | 3e | EXCELLENT | |
25 | ERON DES LOUPS DU RANCH | - | ABSENT |
Chien de berger belge Malinois Classe TRAVAIL MALE Juge : Mme VARLET Marie-France |
26 | EXAU DU MONT DE RICHLOF | 1er | EXCELLENT CACS - CACIB | |
27 | MASKI DU VAN LAPPEENRANTA BANDIT | 2e | EXCELLENT |
Chien de berger belge Malinois Classe INTERMEDIAIRE FEMELLES Juge : Mme VARLET Marie-France |
28 | FIRST-LADY DU MONT DE RICHLOF | 1er | EXCELLENT |
Chien de berger belge Malinois Classe OUVERTE FEMELLES Juge : Mme VARLET Marie-France |
29 | DS DES ILES DU SUD | 2e | EXCELLENT RCACS | |
30 | FARRAH FAWCETT DU DOMAINE DE VAUROUX | - | ABSENT | |
31 | GYPSE DES CHEMINS DU VERDON | 3e | EXCELLENT | |
32 | GYXIE DU MAS DES LAVANDES | 4e | EXCELLENT | |
33 | INFO DU HAMEAU SAINT BLAISE | 1er | EXCELLENT CACS - RCACIB |
Chien de berger belge Malinois Classe CHAMPION FEMELLES Juge : Mme VARLET Marie-France |
34 | EOS DU HAMEAU ST BLAISE | 1er | EXCELLENT CACIB | Meilleur de Race |
Chien de berger belge Malinois Classe JEUNE FEMELLES Juge : Mme VARLET Marie-France |
35 | GALICE DES ILES DU SUD | 3e | EXCELLENT | |
36 | GIN TONIC DE LA VALLEE DU GRAND LOUP | - | DISQUALIFIÉ | |
37 | HADESS DES ILES DU SUD | 1er | EXCELLENT | Meilleur Jeune |
38 | HESTIA DES ILES DU SUD | 2e | EXCELLENT |
Chien de berger belge Tervueren Classe INTERMEDIAIRE MALE Juge : Mme VARLET Marie-France |
39 | FAROUK-T DEI BACI RUBATI | 1er | EXCELLENT RCACS - RCACIB |
Chien de berger belge Tervueren Classe OUVERTE MALE Juge : Mme VARLET Marie-France |
40 | BALOUBET DU PERIGORD VERT | 1er | EXCELLENT CACS - CACIB | |
41 | FIDELIO DU HOME DE LA LOUVE BLANCHE | 2e | EXCELLENT |
Chien de berger belge Tervueren Classe JEUNE MALE Juge : Mme VARLET Marie-France |
42 | GULLIT DU CLOS DU RICHAUMOINE | - | ABSENT |
Chien de berger belge Tervueren Classe INTERMEDIAIRE FEMELLES Juge : Mme VARLET Marie-France |
43 | GAYA DU BLASON DE LA FORGE | 1er | EXCELLENT |
Chien de berger belge Tervueren Classe OUVERTE FEMELLES Juge : Mme VARLET Marie-France |
44 | BUTTERFLY-BIMBO DU HOME DE LA LOUVE BLANCHE | 3e | EXCELLENT | |
45 | COST-AR-C'HOAT DU PUY D'AURA | 2e | EXCELLENT RCACS - RCACIB | |
46 | GUESS DU DOMAINE DE VAUROUX | - | BON | |
47 | VISNJA DE LA FORET D'OLIFAN | 1er | EXCELLENT CACS - CACIB | Meilleur de Race |
Chien de berger belge Tervueren Classe TRAVAIL FEMELLES Juge : Mme VARLET Marie-France |
48 | ALOIS DU MOULIN DU VERGER | 1er | EXCELLENT |
Chien de berger belge Tervueren Classe JEUNE FEMELLES Juge : Mme VARLET Marie-France |
49 | AISCHA VON DEN BELGISCHEN WOLFEN | - | BON | |
50 | GYPSIE DU CLAN MACVAL | 1er | EXCELLENT | Meilleur Jeune |
courtesy: CEDIA
Subscribe to:
Posts (Atom)
NEXT EVENT
15 de Dezembro - 2012 - Póvoa de Varzim